For acquired methemoglobinemia the typical treatment is with methylene blue. This is administered with an IV over a five-minute period and results are typically seen within 20 minutes.
If found early, acquired methemoglobinemia can be easily treated with no side effects. After treatment with methylene blue the patient can expect a full recovery.
Acquired methemoglobinemia can be caused by exposure to certain medications, chemicals, or substances that can oxidize hemoglobin, converting it to methemoglobin. Common causes include nitrate or nitrite medications, certain anesthetics, certain antibiotic drugs (such as dapsone and sulfonamides), and certain chemicals used in industries like aniline dyes or certain pesticides.
Methemoglobinemia is not primarily classified as an X-linked disorder; it is usually caused by genetic mutations that affect the hemoglobin's ability to carry oxygen. The most common forms of hereditary methemoglobinemia are autosomal recessive, linked to mutations in the cytochrome b5 reductase gene. However, certain acquired forms can occur due to exposure to specific chemicals or drugs. While some genetic conditions are X-linked, methemoglobinemia does not fall into that category.
Congenital methemoglobinemia is typically benign and should be observed. If methemoglobinemia symptoms occur the person should be taken to the hospital for treatment.
If a person gets methemoglobinemia from a certain medication that medication should be avoided at all costs in the future. For people with congenital methemoglobinemia medications or other things that are known to oxidize hemoglobin should be avoided.
Water conataining nitrates can cause methemoglobinemia blue baby syndrome). The condition can progress from a blue-grey skin colour to coma and death if not treated..
Methanoglobnemia is mispelled, it's actually methemoglobinemia Methemoglobinemia is a blood disorder in which an abnormal amount of methemoglobin -- a form of hemoglobin -- is produced. Hemoglobin is the molecule in red blood cells that distributes oxygen to the body. Methemoglobin cannot release oxygen. In methemoglobinemia, the hemoglobin is unable to release oxygen effectively to body tissues.
Acquired sideroblastic anemia may be cured when the condition that causes it is treated or removed.
The rate of death among those individuals with acquired brain injury will vary by severity. The death rate will also be based on how one was originally treated.
Bluish-purple skin can be indicative of a genetic disorder known as methemoglobinemia, where an abnormal amount of methemoglobin—a form of hemoglobin that cannot effectively release oxygen to tissues—accumulates in the blood. This condition can be inherited or acquired and results in a characteristic skin discoloration known as cyanosis. Other genetic disorders that might cause similar symptoms could include certain forms of Ehlers-Danlos syndrome or hereditary hemorrhagic telangiectasia, but methemoglobinemia is the most directly associated with bluish-purple skin.
Methemoglobinemia was first described in 1943 by a physicians Alfred Stengel and Albert Mason of the University of Pennsylvania. They identified it as a condition where hemoglobin is unable to carry oxygen effectively due to the presence of excess methemoglobin in the blood.
Yes. An example could be the HIV virus eventually leading to A.I.D.S. if left un-treated.