Yes. Rising glucose levels causes insulin to be secreted.
glucose
The blood-glucose level begins to drop several hours after a meal, leading to a decrease in insulin secretion and a rise in glucagon secretion; glucagon is secreted by the α cells of the pancreas in response to a low blood-sugar level in the fasting state. In same way,insulin signals the fed state, this fed condition leads to the secretion of insulin, which is one of the two most important regulators of fuel metabolism,the secretion of the hormone insulin by the β cells of the pancreas is stimulated by glucose. It stimulates glycogen synthesis in both muscle and the liver and suppresses gluconeogenesis by the liver.
Insulin secretion is primarily regulated by blood glucose levels. When blood glucose levels rise after a meal, beta cells in the pancreas release insulin to help cells uptake glucose for energy production.
Insulin stimulates the uptake of glucose by cells, especially in muscle and fat tissue, which promotes the storage of glucose as glycogen. Insulin also stimulates protein synthesis and inhibits protein breakdown.
Consuming carbohydrates, especially simple sugars, stimulates the release of insulin from the pancreas. Other factors like certain amino acids, fatty acids, and hormones can also trigger insulin secretion. Insulin secretion plays a key role in regulating blood sugar levels by facilitating glucose uptake into cells for energy production.
glucose
Insulin is mainly stimulated by rising blood glucose levels after eating. It is released from the pancreas in response to elevated blood sugar to help regulate glucose uptake by cells, especially muscle and fat cells. Other factors that can stimulate insulin secretion include amino acids from protein-rich foods and certain hormones like incretins.
Beta cells secrete insulin, which lowers blood glucose and stimulates the production of glycogen. Alpha cells secrete glucagon, which raises blood glucose. The secretion of insulin is stimulated by a rise of blood glucose following meals. Glucogon is stimulated by a fall in blood glucose during periods of fasting.
The blood-glucose level begins to drop several hours after a meal, leading to a decrease in insulin secretion and a rise in glucagon secretion; glucagon is secreted by the α cells of the pancreas in response to a low blood-sugar level in the fasting state. In same way,insulin signals the fed state, this fed condition leads to the secretion of insulin, which is one of the two most important regulators of fuel metabolism,the secretion of the hormone insulin by the β cells of the pancreas is stimulated by glucose. It stimulates glycogen synthesis in both muscle and the liver and suppresses gluconeogenesis by the liver.
Insulin secretion is primarily regulated by blood glucose levels. When blood glucose levels rise after a meal, beta cells in the pancreas release insulin to help cells uptake glucose for energy production.
The hyperglycemia caused by stress would lead to an increased secretion of insulin, which would than lower blood glucose. As a consequence, the blood glucose would not be sustained to deal with continued stress.
Glucagons and insulin
Insulin stimulates the uptake of glucose by cells, especially in muscle and fat tissue, which promotes the storage of glucose as glycogen. Insulin also stimulates protein synthesis and inhibits protein breakdown.
Raised glucose levels appear to be due to inhibition of insulin secretion.
Endocrine reflex
Consuming carbohydrates, especially simple sugars, stimulates the release of insulin from the pancreas. Other factors like certain amino acids, fatty acids, and hormones can also trigger insulin secretion. Insulin secretion plays a key role in regulating blood sugar levels by facilitating glucose uptake into cells for energy production.
Insulin is the hormone that allows cells to take up glucose. Insulin is synthesised and released from the beta cells in the islets of langerhans of the pancreas. Its release is stimulated by high blood glucose levels.