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What receptor type is exemplified by opening a channel to let sodium into the cell?
The receptor type exemplified by opening a channel to let sodium into the cell is the ligand-gated ion channel. When a specific ligand, such as a neurotransmitter, binds to the receptor, it causes a conformational change that opens the channel, allowing sodium ions (Na+) to flow into the cell. This influx of sodium typically leads to depolarization of the cell membrane, which can trigger an action potential in neurons. Examples of such receptors include the nicotinic acetylcholine receptor and the glutamate receptor.
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What change does ACh cause in an ACh receptor?
Acetylcholine (ACh) binding to an acetylcholine receptor triggers a conformational change in the receptor protein, leading to the opening of an ion channel within the receptor. This allows specific ions, such as sodium or potassium, to flow across the cell membrane, resulting in changes in membrane potential and ultimately leading to cellular responses.
How are chemically gated channels activated?
Chemically-gated ion channels are receptor membrane proteins that are permeable to specific ions. The 'gating' part of it refers to the channel being open only once activated; which in this case will be by a chemical. An example would be the AMPA glutamate receptor, which has a channel pore that is permeable to sodium ions. Only by binding to glutamate (a neurotransmitter) does the channel allow sodium ions to enter the cell.
Depolarization of the nerve membrane begins with?
The opening of voltage-gated sodium channels in response to a stimulus. Sodium ions flow into the cell, causing depolarization as the inside becomes more positively charged.
What happens when a ligand gated channel is stimulated?
When a ligand-gated channel is stimulated, the specific ligand (or chemical signal) binds to the receptor site on the channel, causing a conformational change in the channel protein. This change opens or closes the channel, allowing specific ions to flow through. This ion movement can generate an electrical signal in the cell and trigger various physiological responses.
Which ion channel opens in response to a change in membrane potential and participates in the generation and conduction of action potentials?
Sodium channels. A neuron's membrane potential may depolarize for many reasons (neurotransmitters, mechanical deflection, electrical synapse, etc). When that membrane depolarizes to the point of its threshold of activation, then voltage gated channels open up an allow an influx of sodium into the cell. This rapidly depolarizes the cell's membrane, causing that upward peak or rising phase to occur.
What change does ACh cause in an ACh receptor?
Acetylcholine (ACh) binding to an acetylcholine receptor triggers a conformational change in the receptor protein, leading to the opening of an ion channel within the receptor. This allows specific ions, such as sodium or potassium, to flow across the cell membrane, resulting in changes in membrane potential and ultimately leading to cellular responses.
What are mechanical gated channels?
Chemically-gated ion channels are receptor membrane proteins that are permeable to specific ions. The 'gating' part of it refers to the channel being open only once activated; which in this case will be by a chemical. An example would be the AMPA glutamate receptor, which has a channel pore that is permeable to sodium ions. Only by binding to glutamate (a neurotransmitter) does the channel allow sodium ions to enter the cell.
How are chemically gated channels activated?
Chemically-gated ion channels are receptor membrane proteins that are permeable to specific ions. The 'gating' part of it refers to the channel being open only once activated; which in this case will be by a chemical. An example would be the AMPA glutamate receptor, which has a channel pore that is permeable to sodium ions. Only by binding to glutamate (a neurotransmitter) does the channel allow sodium ions to enter the cell.
Does receptor proteins pump sodium ions into a cell?
Yes§
A sodium channel blocker would increasedecreaseserum sodium levels outside of normal resting nerve cells?
A sodium channel blocker would decrease serum sodium levels outside of normal resting nerve cells. Sodium channel blockers inhibit the influx of sodium ions into cells, leading to decreased extracellular sodium levels.
Describe the interaction of acetylcholine and calcium ions in stimulating muscle contraction?
The acetylcholine diffuses across the synapse and binds to and activates nicotinic acetylcholine receptors on the motor end plate of the muscle cell. Activation of the nicotinic receptor opens its intrinsic sodium/potassium channel, causing sodium to rush in and potassium to trickle out.
Is a sodium channel that is opened by acetylcholine considered to be voltage-gated?
Yes, a sodium channel that is opened by acetylcholine is not considered to be voltage-gated.
Depolarization of the nerve membrane begins with?
The opening of voltage-gated sodium channels in response to a stimulus. Sodium ions flow into the cell, causing depolarization as the inside becomes more positively charged.
Opening of sodium channels in the membrane of a neuron results in?
depolarization.
What is a sodium ion leak channel?
A sodium ion leak channel is a protein channel in the cell membrane that allows sodium ions to leak into or out of the cell. This helps to maintain the resting membrane potential of the cell by balancing the movement of ions across the membrane. Sodium ion leak channels are important for regulating the excitability of neurons and muscle cells.
What type of ion channel opening is caused by the binding of neurotransmitters to the receptors on the motor endplate?
Sodium enters the cell and causes depolarization. A small amount of potassium also leaves the motor end plate. This means binding of the neurotransmitter causes chemically gated sodium channels to open in the motor end plate.calcium
What type of channel is responsible for the action potential?
sodium potassium and calcium
