Fascicles
The chemical used at the junction with a skeletal muscle is acetylcholine (ACh). It is released from the motor neuron at the neuromuscular junction and binds to receptors on the muscle fiber's membrane, triggering muscle contraction. This process is essential for voluntary movement and is part of the overall neuromuscular signaling mechanism.
ACh (acetylcholine) binds to receptors at the NMJ (neuromuscular junction) to induce contraction of muscle.
tropomyosin moves out of the groove between the actin molecules.
No, it's probably a better way of getting a muscle cramp.
When acetylcholine binds to its receptor in the sarcolemma of a muscle cell, it triggers an action potential to be generated along the muscle cell membrane. This action potential then spreads along the sarcolemma and eventually leads to muscle contraction by initiating the release of calcium ions from the sarcoplasmic reticulum.
chemical A binds with and blocks ACh receptors on muscle cells.
The chemical used at the junction with a skeletal muscle is acetylcholine (ACh). It is released from the motor neuron at the neuromuscular junction and binds to receptors on the muscle fiber's membrane, triggering muscle contraction. This process is essential for voluntary movement and is part of the overall neuromuscular signaling mechanism.
Calmodulin is a protein that binds calcium ions in smooth muscle cells. When calcium binds to calmodulin, it triggers a series of intracellular signaling events that lead to smooth muscle contraction.
Tendons tie muscles to bones and ligaments tie muscle to muscle.
In the bloodsteam, adrenaline acts as a hormone and binds to a few different kinds of adrenoreceptors that are found in skeletal muscle, cardiac muscle, smooth muscle etcDepending on the receptor, it can trigger different cascade pathways that yeild different response for "fightning or fleeing"
It stimulates both receptor with almost the same affinity
Acetylcholine release is necessary for skeletal muscle contraction, because it serves as the first step in the process, enabling the subsequent cross-bridge formation. A muscle's ability to contract depends on the formation of cross-bridges between myosin & actin filaments. A drug that blocks acetylcholine release would interfere with this cross-bridge formation and prevent muscle contraction
Myoglobin is basically a protein that binds oxygen and iron. It is found in the muscle tissue of vertebrates and almost all mammals. It is highly concentrated in skeletal muscles, cardiac muscles and damaged muscle tissues that are similarly known as rhabdomyolysis.
Acetylcholine binds to the muscle cell membrane, causing a temporary rush of calcium ions into the muscle cell. This influx of calcium triggers muscle contraction.
When a muscle cell responds to a stimulus, calcium is released from the sarcoplasmic reticulum. This calcium binds to troponin, triggering a conformational change that allows myosin to bind to actin and initiate muscle contraction.
ACh (acetylcholine) binds to receptors at the NMJ (neuromuscular junction) to induce contraction of muscle.
tropomyosin moves out of the groove between the actin molecules.