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When presynaptic cells produce action potentials, it triggers the opening of voltage-gated calcium channels in the presynaptic membrane. This influx of calcium ions into the presynaptic cell triggers the release of neurotransmitter molecules from small, membrane-bound vesicles. The released neurotransmitters then diffuse across the synapse and bind to receptors on the postsynaptic cell, generating a response in the postsynaptic cell.
A neuron (nerve cell) receives dendritic input in order to generate action potentials to transmit signals of the same. After the action potential triggers release of neurotransmitters in the axonal terminal of that neuron, those neurotransmitters propagate the signal forward to the next neuron, and so forth.
Every time neurotransmitter is released from the presynaptic neuron it generates an excitatory post synaptic potential(EPSP) in the postsynaptic neuron. When the EPSP is greater than the threshold for excitation an action potential is generated.
Your question isn't very clear.... Presynaptic knob is the neurone before the synapse. Postsynaptic knob is the neurone after the synapse. Calcium ions diffuse into the presynaptic knob down their concentration gradient when an impulse arrives at the presynaptic knob. This causes the vesicles to move towards the presynaptic membrane and fuse with it. This releases the neurotransmitter (e.g. Ach). The Ach diffuses down their concentration gradient in the synaptic cleft then binds with receptors on the post synaptic membrane. This binding causes the Na+ ion channels to open, and the influx of Na+ ions causes depolarisation, and a new action potential in the postsynaptic knob. Then the acetate and choline diffuses back into the presynaptic membrane and is recombined using ATP.
A synaptic potential exists at the INPUT of a neuron (dendrite), and an action potential occurs at the OUTPUT of a neuron (axon). (from OldGuy)(from Ilantoren:) A synaptic potential is the result of many excitatory post synaptic potentials (epsp) each one caused by the synaptic vesicles released by the pre-synaptic terminus. If there are enough of these epsp then the responses will summate and depolarize the post-synaptic membrane at the axon hillock enough to fire an action potential.
Presynaptic neurons release the neurotransmitter in response to an action potential. Postsynaptic neurons receive the neurotransmitter (and can however become presynaptic to the next nerve cell, if the neurotransmitter has stimulated the cell enough).
When presynaptic cells produce action potentials, it triggers the opening of voltage-gated calcium channels in the presynaptic membrane. This influx of calcium ions into the presynaptic cell triggers the release of neurotransmitter molecules from small, membrane-bound vesicles. The released neurotransmitters then diffuse across the synapse and bind to receptors on the postsynaptic cell, generating a response in the postsynaptic cell.
A neuron (nerve cell) receives dendritic input in order to generate action potentials to transmit signals of the same. After the action potential triggers release of neurotransmitters in the axonal terminal of that neuron, those neurotransmitters propagate the signal forward to the next neuron, and so forth.
Let's picture a presynaptic neuron, a synaptic cleft, and a postsynaptic neuron. An action potential reaches the terminal of a presynaptic neurone and triggers an opening of Ca ions enters into the depolarized terminal. This influx of Ca ions causes the presynaptic vesicles to fuse with the presynaptic membrane. This releases the neurotransmitters into the synaptic cleft. The neurotransmitters diffuse through the synaptic cleft and bind to specific postsynaptic membrane receptors. This binding changes the receptors into a ion channel that allows cations like Na to enter into the postsynaptic neuron. As Na enters the postsynaptic membrane, it begins to depolarize and an action potential is generated.
Every time neurotransmitter is released from the presynaptic neuron it generates an excitatory post synaptic potential(EPSP) in the postsynaptic neuron. When the EPSP is greater than the threshold for excitation an action potential is generated.
This can be caused by inhibitor molecules known as neurotoxins occupying the active sites of the receptor molecules of the postsynaptic neurone membrane, this prevents an action potential from being carried from the presynaptic neurone to the postsynaptic neurone, thus preventing the passage of a nerve impulse and consequental muscular contractions that produce an effect to stimuli
The synaptic transmission is where the communication between the terminal button and the dendrite occur. What happens is the impulse moves along the axon and release neurotransmitter from the end plate of the presynaptic neuron and are diffused across the synaptic cleft. This creates a depolarization of the dendrites of the postsynaptic neuron. When that happens the postsynaptic's sodium channels to open and start the action potential. Once the channels are open an enzyme called cholinesterase is released from postsynaptic membrane and it acts to destroy the neurotransmitters. When they are destroyed the sodium channels close and begins recovery.
End plate potential is the change in potential from neurotransmitters. It can be excitatory or inhibitory. If the action potential wants to continue, it will be excitatory and vice versa. It can be additive, if more action potentials are fired it will increase the end plate potential. An action potential is an all or none response. It will either proceed or it will not proceed depending on the terms of the threshold. It cannot be additive, because there is an absolute refractory period where no additional action potentials can be fired.
An excitatory postsynaptic potential, a type of graded potential, occurs because of the influx of Na+ through chemically gated channels in the receptive region, or postsynaptic membrane, of a neuron. Graded potentials are generated by chemically gated channels, whereas action potentials are produced by voltage-gated channels.
Your question isn't very clear.... Presynaptic knob is the neurone before the synapse. Postsynaptic knob is the neurone after the synapse. Calcium ions diffuse into the presynaptic knob down their concentration gradient when an impulse arrives at the presynaptic knob. This causes the vesicles to move towards the presynaptic membrane and fuse with it. This releases the neurotransmitter (e.g. Ach). The Ach diffuses down their concentration gradient in the synaptic cleft then binds with receptors on the post synaptic membrane. This binding causes the Na+ ion channels to open, and the influx of Na+ ions causes depolarisation, and a new action potential in the postsynaptic knob. Then the acetate and choline diffuses back into the presynaptic membrane and is recombined using ATP.
A synaptic potential exists at the INPUT of a neuron (dendrite), and an action potential occurs at the OUTPUT of a neuron (axon). (from OldGuy)(from Ilantoren:) A synaptic potential is the result of many excitatory post synaptic potentials (epsp) each one caused by the synaptic vesicles released by the pre-synaptic terminus. If there are enough of these epsp then the responses will summate and depolarize the post-synaptic membrane at the axon hillock enough to fire an action potential.
When the action potential reaches the button(axon terminal) of the presynaptic neuron the depolarization causes voltage gated calcium channels to open increasing intracellular calcium content. This causes synaptic vesicles to fuse to the membrane and release neurotransmitters that bind to the post synaptic neuron and create a chemical action potential.