0
All I know is that acetylcholine is the only neurotransmitter that transmits signals from nerves to skeletal muscles, so anything that blocks it would probably have the effect of you wanting to move your muscles, but either not being able to, or moving weakly.
Just a (educated) guess.
It would cause spastic paralysis (muscles are contracted and unable to relax)
What else can I help you with?
How would the loss of acetylcholinesterase from the motor end affect skeletal muscle?
Acetylcholine release is necessary for skeletal muscle contraction, because it serves as the first step in the process, enabling the subsequent cross-bridge formation. A muscle's ability to contract depends on the formation of cross-bridges between myosin & actin filaments. A drug that blocks acetylcholine release would interfere with this cross-bridge formation and prevent muscle contraction
While Botulinum toxin blocks the release of acetylcholine the tetanoid toxin actually blocks the release of GABA. How would the effects of tetanoid toxin exposure be different from the results of BT?
The blocking of GABA release will cause convulsions, where as the blocking of acetylcholine will cause paralysis.
How does blocking the activity of acetylcholinesterase affect skeletal muscle?
Acetylcholine release is necessary for skeletal muscle contraction, because it serves as the first step in the process, enabling the subsequent cross-bridge formation. A muscle's ability to contract depends on the formation of cross-bridges between myosin & actin filaments. A drug that blocks acetylcholine release would interfere with this cross-bridge formation and prevent muscle contraction
The botulinus toxin that causes botulism acts by?
Botulinus toxin blocks the release of acetylcholine from the presynaptic terminal. This is a total blockage of neuronal transmission.
Botulin blocks the release of ACh. Botulin is best described as a(n)?
Botulin is best described as a neurotoxin that inhibits the release of acetylcholine (ACh) at the neuromuscular junction, leading to muscle paralysis.
Does atropine block only nicotinic receptors?
Atropine does not only block nicotinic receptors but also acetylcholine at muscarinic receptors
What is a drug that blocks the release of neurotransmitters?
Botulinum toxin, commonly known as Botox, is a drug that blocks the release of acetylcholine at the neuromuscular junction, preventing muscle contractions. This action can help to reduce muscle spasticity, dystonia, and other conditions related to excessive muscle activity.
A drug that decreases peristalsis would be one that?
A drug that decreases peristalsis would likely be an anticholinergic medication, which blocks the action of acetylcholine in the gastrointestinal tract. This inhibition can slow down the contractions of the smooth muscle in the intestines, reducing peristalsis.
What blocks the action of acetylcholine?
Acetylcholine Inhibitors...That would include organophosphates, curare (or tubocuranine), succinylcholine, (a flaccid paralytic used in anesthesia to relax striated muscles for intubation), and various neurotoxins.
What is the effect of curare on eliciting an action potential?
it is an alpha-toxin that binds to acetylcholine binding sites on the postsynaptic cell membrane, which prevents the acetylcholine from acting. Curare blocks synaptic transmission by preventing neural impulses to flow from neuron to neuron. It does allow the action potential to travel in the axon, it just doesn't pass it on to the dendrite.
What is the effect of atropine plus acetylcholine on frog heart?
ACh will decrease heart rate/contractile strength, etc. Atropine is a muscarinic ACh receptor (mAChR) antagonist, so blocks the effects of ACh. Adding both together will result in a weak action of ACh that tails off as all the mAChRs become blocked by Atropine.
What effect does Curare have on muscle tissue?
It blocks the nicotinic cholinergic receptors on the muscle that normally bind the acetylcholine released by the motor neuron.
