Tay-Sach's has a defective gene on chromosme 15

codominant alleles

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50-50. Each parent contributes one pair of genes to their offspring. HD is carried on a Dominant gene. If your father has HD, you may inherit the recessive gene (No HD) or the dominant gene (HD). Odds are exactly even.

Sadly, the prognosis for a child with classic Tay-Sachs disease is certain death. Because the chronic form of Tay-Sachs has been discovered recently, prognosis for this type of the disease is not completely known.

Yes.

A person with tay sachs can live a healthy life but still battles the many limitations of Tay sachs disease. Depending on the type of tay sachs, Classic, Juvenile onset, and Late Onset depends how healthy a life a person with tay sachs disease.

A mutation of the HEX A gene of chromosome 15 causes Tay-Sachs disease. As a result, the protein hexosaminidase A is not formed properly and GM2 ganglioside, the lipid normally broken down by hexosaminidase A, accumulates to toxic levels (especially in the brain).

Almost no kids get Tay - Sachs disease. Of the kids who do get it, they are mostly Jewish, since that is where the disease is most prevelent, and most Jewish people don't intermarry. About one in every 250 people are carriers.

There are three forms of Tay-Sachs disease, categorized by the types of symptoms and the age when the symptoms first appear. The most common form appears when the child is 3 to 6 months old, with the disease progressing rapidly to death by age 4 or 5. At birth, the infant appears healthy and develops normally for the first few months of life, but as the buildup of ganglioside GM2 begins to affect nerves, symptoms appear.

Initially, symptoms may include:

* Seizures

* Noticeable behavior changes, such as the infant stops smiling, crawling or rolling over and loses the ability to grasp or reach out

* Increased startle reaction

* Decreased eye contact

* Listlessness

* Increasing irritability

* Slow body growth with increasing head size

* Delayed mental and social skills

As the disease progresses, these symptoms become more dominant:

* Feeding difficulties

* Abnormal body tone

* Loss of motor skills

* Blindness

* Deafness

* Loss of intellectual skills

Much rarer are the forms of Tay-Sachs that develop later in life and are due to low levels of the hex A enzyme, rather than the complete deficiency of hex A in the infantile form. Children with juvenile hex A deficiency develop symptoms between ages 2 and 5, and usually die by age 15. A milder form of hex A deficiency can develop anywhere from age 5 to the early 30s. Symptoms include slurred speech, an unsteady gait, tremors and, sometimes, mental illness.

lysosomes

It is unknown how many people have Tay-Sachs disease because sometimes the illness is misdiagnosed or not diagnosed in patients. However, it is a very rare disorder that is more common for people with an eastern and central European background.

Lysosomes

chromosome 17 The correct answer is chromosome 15

Brazil

No, never.

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If you have a history with the disease it is verry likely you may bing back to it.

Sachs Elan was created in 1995.